CRISPR-Cas9 correction of OPA1 c.1334G>A: p.R445H restores
Por um escritor misterioso
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Understanding the molecular basis and pathogenesis of hereditary
Idebenone fails to significantly alter dendritic complexity in
Retinal Ganglion Cells in a Dish: Current Strategies and
Homology directed correction, a new pathway model for point
CRISPR-Cas9 correction of OPA1 c.1334G>A: p.R445H restores
CRISPR-Cas9 correction of OPA1 c.1334G>A: p.R445H restores
CRISPR-Cas9 correction of OPA1 c.1334G>A: p.R445H restores
Drosophila model to clarify the pathological significance of OPA1
Optic Atrophy 1 Controls Human Neuronal Development by Preventing
IJMS, Free Full-Text
Drosophila model to clarify the pathological significance of OPA1
JCM, Free Full-Text
Frontiers Establishing induced pluripotent stem cell lines from
Precise gene deletion and replacement using the CRISPR/Cas9 system
OPA1-Exon4b is not required for mitochondrial fusion but ensues
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por adulto (o preço varia de acordo com o tamanho do grupo)